|
Title |
Professor |
|
Contact information |
|
|
External Link |
|
Research Areas
-
Life Science / Genetics
-
Life Science / Psychiatry
-
Humanities & Social Sciences / Special needs education
External Career
-
名古屋工業大学 保健センター センター長・学生なんでも相談室 障害学生支援部門長
2022.04
-
Nagoya Institute of Technology Professor
2021.05
-
Nagoya University Nagoya University Hospital Psychiatry for Parents and Children Assistant Professor
2017.04 - 2021.04
Professional Memberships
-
THE JAPAN SOCIETY OF HUMAN GENETICS
-
THE JAPANESE SOCIETY OF SLEEP RESEARCH
-
日本精神神経学会/専門医・指導医
-
The Japanese Society for Child and Adolescent Psychiatry
Papers
-
Exome sequencing analysis of Japanese autism spectrum disorder case-control sample supports an increased burden of synaptic function-related genes Reviewed
Hiroki Kimura, Masahiro Nakatochi, Branko Aleksic, James Guevara, Miho Toyama, Yu Hayashi, Hidekazu Kato, Itaru Kushima, Mako Morikawa, Kanako Ishizuka, Takashi Okada, Yoshinori Tsurusaki, Atsushi Fujita, Noriko Miyake, Tomoo Ogi, Atsushi Takata, Naomichi Matsumoto, Joseph Buxbaum, Norio Ozaki, Jonathan Sebat
Translational Psychiatry 12 ( 1 ) 2022.12
Publishing type：Research paper (scientific journal) Publisher：Springer Science and Business Media LLC
Abstract
Autism spectrum disorder (ASD) is a highly heritable, complex disorder in which rare variants contribute significantly to disease risk. Although many genes have been associated with ASD, there have been few genetic studies of ASD in the Japanese population. In whole exomes from a Japanese ASD sample of 309 cases and 299 controls, rare variants were associated with ASD within specific neurodevelopmental gene sets, including highly constrained genes, fragile X mental retardation protein target genes, and genes involved in synaptic function, with the strongest enrichment in trans-synaptic signaling (p = 4.4 × 10<sup>−4</sup>, Q-value = 0.06). In particular, we strengthen the evidence regarding the role of ABCA13, a synaptic function-related gene, in Japanese ASD. The overall results of this case-control exome study showed that rare variants related to synaptic function are associated with ASD susceptibility in the Japanese population.DOI： 10.1038/s41398-022-02033-6
Other Link： https://www.nature.com/articles/s41398-022-02033-6
-
A systematic review and network meta-analysis of antimanic drugs for the treatment of acute mania used in Japan Reviewed
Kanako Ishizuka, Toshiya Inada
Psychiatry and Clinical Neurosciences Reports 2022.12
Authorship：Lead author Language：English
-
Exome sequencing of Japanese schizophrenia multiplex families supports the involvement of calcium ion channels Reviewed
Miho Toyama, Yuto Takasaki, Aleksic Branko, Hiroki Kimura, Hidekazu Kato, Yoshihiro Nawa, Itaru Kushima, Kanako Ishizuka, Teppei Shimamura, Tomoo Ogi, Norio Ozaki
PLOS ONE 17 ( 5 ) e0268321 - e0268321 2022.05
Publishing type：Research paper (scientific journal) Publisher：Public Library of Science (PLoS)
Background
Most sequencing studies of schizophrenia (SCZ) have focused on de novo genetic variants due to interpretability. However, investigating shared rare variants among patients in the same multiplex family is also important. Relatively large-scale analyses of SCZ multiplex families have been done in Caucasian populations, but whether detected variants are also pathogenic in the Japanese population is unclear because of ethnic differences in rare variants.
Materials and methods
We performed whole-exome sequencing (WES) of 14 Japanese SCZ multiplex families. After quality control and filtering, we identified rare variants shared among affected persons within the same family. A gene ontology (GO) analysis was performed to identify gene categories possibly affected by these candidate variants.
Results
We found 530 variants in 486 genes as potential candidate variants from the 14 SCZ multiplex families examined. The GO analysis demonstrated significant enrichment in calcium channel activity.
Conclusion
This study provides supporting evidence that calcium ion channel activity is involved in SCZ. WES of multiplex families is a potential means of identifying disease-associated rare variants for SCZ. -
Investigation of OLIG2 as a candidate gene for schizophrenia and autism spectrum disorder. Reviewed
Sho Furuta, Branko Aleksic, Yoshihiro Nawa, Hiroki Kimura, Itaru Kushima, Kanako Ishizuka, Hidekazu Kato, Miho Toyama, Yuko Arioka, Daisuke Mori, Mako Morikawa, Toshiya Inada, Norio Ozaki
Nagoya journal of medical science 84 ( 2 ) 260 - 268 2022.05
Language：English Publishing type：Research paper (scientific journal)
A number of genomic mutations that are thought to be strongly involved in the development of schizophrenia (SCZ) and autism spectrum disorder (ASD) have been identified. Abnormalities involving oligodendrocytes have been reported in SCZ, and as a related gene, oligodendrocyte lineage transcription factor 2 (OLIG2) has been reported to be strongly associated with SCZ. In this study, based on the common disease-rare variant hypothesis, target sequencing of candidate genes was performed to identify rare mutations with a high effect size and the possibility that the identified mutations may increase the risks of SCZ and ASD in the Japanese population. In this study, the exon region of OLIG2 was targeted; 370 patients with SCZ and 192 with ASD were subjected to next-generation sequencing. As a result, one rare missense mutation (A33T) was detected. We used the Sanger method to validate this missense mutation with a low frequency (<1%), and then carried out a genetic association analysis involving 3299 unrelated individuals (1447 with SCZ, 380 with ASD, and 1472 healthy controls) to clarify whether A33T was associated with SCZ or ASD. A33T was not found in either case group, and in only one control. We did not find evidence that p.A33T is involved in the onset of ASD or SCZ; however, associations with this variant need to be evaluated in larger samples to confirm our results.
-
Psychiatric patients with a de novo 17q12 deletion: two case reports. Reviewed International journal
Itaru Kushima, Mariko Uematsu, Kanako Ishizuka, Branko Aleksic, Norio Ozaki
Psychiatry and clinical neurosciences 2022.04
-
Cross-disorder analysis of genic and regulatory copy number variations in bipolar disorder, schizophrenia, and autism spectrum disorder Reviewed
Itaru Kushima, Masahiro Nakatochi, Branko Aleksic, Takashi Okada, Hiroki Kimura, Hidekazu Kato, Mako Morikawa, Toshiya Inada, Kanako Ishizuka, Youta Torii, Yukako Nakamura, Satoshi Tanaka, Miho Imaeda, Nagahide Takahashi, Maeri Yamamoto, Kunihiro Iwamoto, Yoshihiro Nawa, Nanayo Ogawa, Shuji Iritani, Yu Hayashi, Tzuyao Lo, Gantsooj Otgonbayar, Sho Furuta, Nakao Iwata, Masashi Ikeda, Takeo Saito, Kohei Ninomiya, Tomo Okochi, Ryota Hashimoto, Hidenaga Yamamori, Yuka Yasuda, Michiko Fujimoto, Kenichiro Miura, Masanari Itokawa, Makoto Arai, Mitsuhiro Miyashita, Kazuya Toriumi, Kazutaka Ohi, Toshiki Shioiri, Kiyoyuki Kitaichi, Toshiyuki Someya, Yuichiro Watanabe, Jun Egawa, Tsutomu Takahashi, Michio Suzuki, Tsukasa Sasaki, Mamoru Tochigi, Fumichika Nishimura, Hidenori Yamasue, Hitoshi Kuwabara, Tomoyasu Wakuda, Takahiro A. Kato, Shigenobu Kanba, Hideki Horikawa, Masahide Usami, Masaki Kodaira, Kyota Watanabe, Takeo Yoshikawa, Tomoko Toyota, Shigeru Yokoyama, Toshio Munesue, Ryo Kimura, Yasuko Funabiki, Hirotaka Kosaka, Minyoung Jung, Kiyoto Kasai, Tempei Ikegame, Seiichiro Jinde, Shusuke Numata, Makoto Kinoshita, Tadafumi Kato, Chihiro Kakiuchi, Kazuhiro Yamakawa, Toshimitsu Suzuki, Naoki Hashimoto, Shuhei Ishikawa, Bun Yamagata, Shintaro Nio, Toshiya Murai, Shuraku Son, Yasuto Kunii, Hirooki Yabe, Masumi Inagaki, Yu-ichi Goto, Yuto Okumura, Tomoya Ito, Yuko Arioka, Daisuke Mori, Norio Ozaki
Biological Psychiatry 92 ( 5 ) 362 - 374 2022.04
Publishing type：Research paper (scientific journal) Publisher：Elsevier BV
-
Autistic traits as predictors of persistent depression. Reviewed International journal
Kanako Ishizuka, Tomomi Ishiguro, Norio Nomura, Toshiya Inada
European archives of psychiatry and clinical neuroscience 272 ( 2 ) 211 - 216 2022.03
Authorship：Lead author Language：English Publishing type：Research paper (scientific journal)
Persistent depression has been suggested to be associated with autistic traits in people of working age. This study aimed to clarify which autistic characteristics at the initial visit were associated with persistent depression at the 12 week follow-up in a primary care setting. Newly depressed outpatients aged 24-59 years with no history of autism were asked to complete the 50-item autism spectrum quotient (AQ) and the Beck depression inventory (BDI) at baseline and 12 week follow-up (N = 123, males: 48%, age: 37.7 ± 9.15 years). Nearly 40% of participants had an AQ score ≥ 26. Significant differences were observed between the group with remitted depression (N = 43) and those with persistent depression (N = 80) in educational years and AQ "attention switching" and "attention to detail" subscale scores. In addition, a statistically significant decrease in the total AQ and the "communication" and "imagination" scores were observed in the remitted group, while no such change was observed in the group with persistent depression. It remains unclear whether the self-perceived severity of communication and imagination traits in persistent depression was due to the state of persistent depression or a kind of premorbid autistic trait. The results suggest that high levels of autistic traits are frequently present in adults with depression. High "attention switching" and "attention to detail" scores in AQ screening at the first visit might predict the persistence of depressive symptoms after 12 weeks in adults with depression, while total AQ scores, especially for "communication" and "imagination" items, might be state-dependent.
-
Identification of ultra-rare disruptive variants in voltage-gated calcium channel-encoding genes in Japanese samples of schizophrenia and autism spectrum disorder. Reviewed International journal
Chenyao Wang, Shin-Ichiro Horigane, Minoru Wakamori, Shuhei Ueda, Takeshi Kawabata, Hajime Fujii, Itaru Kushima, Hiroki Kimura, Kanako Ishizuka, Yukako Nakamura, Yoshimi Iwayama, Masashi Ikeda, Nakao Iwata, Takashi Okada, Branko Aleksic, Daisuke Mori, Takashi Yoshida, Haruhiko Bito, Takeo Yoshikawa, Sayaka Takemoto-Kimura, Norio Ozaki
Translational psychiatry 12 ( 1 ) 84 - 84 2022.02
Language：English Publishing type：Research paper (scientific journal)
Several large-scale whole-exome sequencing studies in patients with schizophrenia (SCZ) and autism spectrum disorder (ASD) have identified rare variants with modest or strong effect size as genetic risk factors. Dysregulation of cellular calcium homeostasis might be involved in SCZ/ASD pathogenesis, and genes encoding L-type voltage-gated calcium channel (VGCC) subunits Cav1.1 (CACNA1S), Cav1.2 (CACNA1C), Cav1.3 (CACNA1D), and T-type VGCC subunit Cav3.3 (CACNA1I) recently were identified as risk loci for psychiatric disorders. We performed a screening study, using the Ion Torrent Personal Genome Machine (PGM), of exon regions of these four candidate genes (CACNA1C, CACNA1D, CACNA1S, CACNA1I) in 370 Japanese patients with SCZ and 192 with ASD. Variant filtering was applied to identify biologically relevant mutations that were not registered in the dbSNP database or that have a minor allele frequency of less than 1% in East-Asian samples from databases; and are potentially disruptive, including nonsense, frameshift, canonical splicing site single nucleotide variants (SNVs), and non-synonymous SNVs predicted as damaging by five different in silico analyses. Each of these filtered mutations were confirmed by Sanger sequencing. If parental samples were available, segregation analysis was employed for measuring the inheritance pattern. Using our filter, we discovered one nonsense SNV (p.C1451* in CACNA1D), one de novo SNV (p.A36V in CACNA1C), one rare short deletion (p.E1675del in CACNA1D), and 14 NSstrict SNVs (non-synonymous SNV predicted as damaging by all of five in silico analyses). Neither p.A36V in CACNA1C nor p.C1451* in CACNA1D were found in 1871 SCZ cases, 380 ASD cases, or 1916 healthy controls in the independent sample set, suggesting that these SNVs might be ultra-rare SNVs in the Japanese population. The neuronal splicing isoform of Cav1.2 with the p.A36V mutation, discovered in the present study, showed reduced Ca2+-dependent inhibition, resulting in excessive Ca2+ entry through the mutant channel. These results suggested that this de novo SNV in CACNA1C might predispose to SCZ by affecting Ca2+ homeostasis. Thus, our analysis successfully identified several ultra-rare and potentially disruptive gene variants, lending partial support to the hypothesis that VGCC-encoding genes may contribute to the risk of SCZ/ASD.
-
Autism spectrum disorder comorbid with obsessive compulsive disorder and eating disorder in a woman with NBEA deletion. Reviewed International journal
Hidekazu Kato, Itaru Kushima, Akira Yoshimi, Kanako Ishizuka, Hiroki Kimura, Branko Aleksic, Nagahide Takahashi, Takashi Okada, Norio Ozaki
Psychiatry and clinical neurosciences 76 ( 1 ) 36 - 38 2022.01
-
Catatonia and Delirium: Similarity and Overlap of Acute Brain Dysfunction. Invited Reviewed International journal
Masako Tachibana, Kanako Ishizuka, Toshiya Inada
Frontiers in psychiatry 13 876727 - 876727 2022
Language：English Publishing type：Research paper (scientific journal)
Books and Other Publications
-
知的能力障害／今日の診断指針第8版
石塚佳奈子（ Role： Contributor）
医学書院 2020.03 （ ISBN:9784260038089 ）
Total pages：xxvii, 2079p Language：jpn
-
神経発達症／メディカルスタッフ専門基礎科目シリーズ精神医学（飯高哲也編著）
石塚佳奈子（ Role： Contributor）
理工図書 2018.08
Responsible for pages：281-309 Language：jpn Book type：Scholarly book
-
乳幼児期の診断／乳幼児精神保健の基礎と実践（青木豊，松本英夫編）
石塚佳奈子, 本城秀次（ Role： Contributor）
岩崎学術出版社 2017.06
Responsible for pages：140-148 Language：jpn Book type：Scholarly book
-
精神疾患の遺伝を患者家族とどう話し合うか／最新精神・神経遺伝医学研究と遺伝カウンセリング（戸田達史編）
石塚佳奈子, 尾崎紀夫（ Role： Contributor）
メディカル ドゥ 2017.04
Responsible for pages：255-259 Language：jpn Book type：Scholarly book
-
22q11.2欠失症候群／精神医学症候群（第2版）
石塚佳奈子, 尾崎紀夫（ Role： Contributor）
日本臨牀 2017.03
Responsible for pages：357-362 Language：jpn Book type：Scholarly book
-
物質使用障害／臨床児童青年精神医学ハンドブック（本城秀次，野邑健二，岡田俊編）
石塚佳奈子（ Role： Contributor）
西村書店 2016.11
Responsible for pages：312-318 Language：jpn
-
選択性緘黙／今日の精神疾患治療指針第2版（樋口輝彦，市川宏伸，神庭重信，朝田隆，中込和幸編）
石塚佳奈子, 本城秀次（ Role： Contributor）
医学書院 2016.10
Responsible for pages：194-196 Language：jpn Book type：Scholarly book
-
統合失調症／こどもの病気 遺伝について聞かれたら（松原洋一，呉繁夫，左合治彦編）
石塚佳奈子, 宍戸恵美子, 尾崎紀夫（ Role： Contributor）
診断と治療社 2015.03
Responsible for pages：147-148 Language：jpn Book type：Scholarly book
-
うつ病，双極性障害／こどもの病気 遺伝について聞かれたら（松原洋一，呉繁夫，左合治彦編）
石塚佳奈子, 宍戸恵美子, 尾崎紀夫（ Role： Contributor）
診断と治療社 2015.03
Responsible for pages：144-146 Language：jpn Book type：Scholarly book
-
分離不安症／DSM-5を読み解く4（神庭重信, 三村 將編）
石塚佳奈子（ Role： Contributor）
中山書店 2014.11
Responsible for pages：18-25 Language：jpn Book type：Scholarly book
Misc
-
本邦における統合失調症と自閉スペクトラム症に関連するGRIN2Bの遺伝子変異の探索
高崎 悠登, 尾崎 紀夫, 小出 隆義, 王 晨堯, 木村 大樹, 久島 周, 石塚 佳奈子, 森 大輔, 池田 匡志, アレクシッチ・ブランコ, 岡田 俊, 江川 純, 桑原 斉, 染矢 俊幸, 吉川 武男, 岩田 仲生
精神神経学雑誌 ( 2017特別号 ) S626 - S626 2017.06
Language：Japanese Publisher：(公社)日本精神神経学会
-
統合失調症発症に強い影響を及ぼす、頻度の低い稀な遺伝子変異を22q11.2欠失領域に存在するミエリン関連遺伝子のRTN4Rに同定した
木村 大樹, 尾崎 紀夫, 藤田 幸, 川端 猛, 石塚 佳奈子, Wang Chenyao, 岩山 佳美, 岡久 祐子, 久島 周, 森川 真子, 宇野 洋太, 岡田 俊, 森 大輔, 池田 匡志, 稲田 俊也, Aleksic Branko, 吉川 武男, 岩田 仲生, 中村 春木, 山下 俊英
( 2017特別号 ) S622 - S622 2017.06
Language：Japanese Publisher：(公社)日本精神神経学会
Presentations
-
困難を抱える児童生徒への個別支援に関する、大学生の許容度および知識獲得の影響
石塚佳奈子
第63回日本児童青年精神医学会総会 2022.11
Event date： 2022.11
Language：Japanese Presentation type：Poster presentation
Venue：長野県
-
評価尺度を用いた抑うつ症状の重症度評価を行う際の留意点 大人のADHDにおける抑うつ症状の評価
石塚 佳奈子
第117回日本精神神経学会学術総会 ワークショップ 2021.09
Event date： 2021.09
-
当事者・家族と進めるゲノム医療へ ゲノム解析・遺伝カウンセリングに携わる臨床遺伝専門医の立場から
石塚 佳奈子
第116回日本精神神経学会学術総会 倫理委員会シンポジウム 2020.09
Event date： 2020.09
Language：Japanese Presentation type：Symposium, workshop panel (nominated)
-
日常臨床で話題になる家族の発症リスク
石塚 佳奈子, 尾崎 紀夫
日本児童青年精神医学会総会抄録集 2019.12 (一社)日本児童青年精神医学会
Event date： 2019.12
Language：Japanese
-
小児の社会機能低下を親の抑うつから読み解く
石塚 佳奈子
メンタルヘルス岡本記念財団研究助成報告集 2019.03 (公財)メンタルヘルス岡本記念財団
Event date： 2019.03
Language：Japanese
小児の社会機能低下に及ぼす親の抑うつの影響を明らかにすることを目的に、児童精神科外来を受診した児10症例(男児3例、女児7例、平均年齢11.7±2.2歳)とその親を対象に、児の社会機能については社会的職業的機能評定尺度の構造化評価システムを、親の抑うつについてはベック抑うつ質問票(BDI)をそれぞれ用いて評価した。その結果、親の抑うつについてはBDI 31点以上の重度のうつ状態、もしくは30分以上の時間をかけても全21項目への回答が選択できない状態が多く見られた。今回の結果から、小児の社会機能低下と親の抑うつは必ずしも関連付けられない可能性が示唆され、小児期以前の乳幼児期からの継続的な社会機能と親の抑うつの関連を明らかにする必要があると考えられた。
-
成長曲線の観点から考察した神経性やせ症の1例
松田 慶子, 石塚 佳奈子, 尾崎 紀夫
日本児童青年精神医学会総会抄録集 2018.10 (一社)日本児童青年精神医学会
Event date： 2018.10
Language：Japanese
-
あえて遺伝の話をしよう
石塚 佳奈子, 尾崎 紀夫
日本児童青年精神医学会総会抄録集 2018.10 (一社)日本児童青年精神医学会
Event date： 2018.10
Language：Japanese
-
MUTATION SCREENING OF THE PCDH15 GENE IN PATIENTS SUFFERING FROM AUTISM SPECTRUM DISORDERS AND SCHIZOPHRENIA
Ishizuka Kanako, Wang Chenyao, Kimura Hiroki, Xing Jingrui, Kushima Itaru, Arioka Yuko, Yoshimi Akira, Nakamura Yukako, Oya-Ito Tomoko, Takasaki Yuto, Uno Yota, Okada Takashi, Mori Daisuke, Aleksic Branko, Ozaki Norio
EUROPEAN NEUROPSYCHOPHARMACOLOGY 2017.10
Event date： 2017.10
Language：English
-
児童青年期の重篤な精神症状を経て良好な経過をたどる成人2症例の考察
石塚 佳奈子, 岡田 俊, 尾崎 紀夫
日本児童青年精神医学会総会抄録集 2016.10 (一社)日本児童青年精神医学会
Event date： 2016.10
Language：Japanese
-
22q11.2重複を有する神経発達症症例の1考察
石塚 佳奈子, 久島 周, アレクシッチ・ブランコ, 尾崎 紀夫
日本神経精神薬理学会年会プログラム・抄録集 2016.07 (一社)日本神経精神薬理学会
Event date： 2016.07
Language：Japanese
Awards
-
2017年度国際学会発表奨励賞（後期）
2018 日本生物学的精神医学会 Investigation of novel rare variants in NRXN1 contributes to the increased risk of autism spectrum disorders and schizophrenia
石塚佳奈子
Award type：Award from international society, conference, symposium, etc. Country：Japan
-
名古屋大学総長顕彰
2003 名古屋大学
Country：Japan
Scientific Research Funds Acquisition Results
-
併存する不安症を起点とした自閉スペクトラム症の新たな病態解明
Grant number：20K16625 2020.04 - 2023.03
日本学術振興会 科学研究費助成事業 若手研究 若手研究
石塚 佳奈子
Authorship：Principal investigator
Grant amount：\4160000 （ Direct Cost: \3200000 、 Indirect Cost：\960000 ）
-
AYA世代の精神疾患高リスク群における予防的睡眠マネジメントに関する研究
2020.04 - 2021.04
国立研究開発法人日本医療研究開発機構（AMED） 令和2年度 障害者対策総合研究開発事業
石塚佳奈子(分担)
Authorship：Coinvestigator(s)
-
Genotype-to-phenotypeによる精神障害の新たな病態解明
2018.04 - 2020.03
日本学術振興会 科学研究費助成事業 若手研究
石塚佳奈子
Authorship：Principal investigator Grant type：Competitive
-
NRXN1を起点に自閉スペクトラム症と統合失調症をgenotype-to-phenotypeで再構築する試み
2018.04 - 2019.03
川野正登記念（公財）川野小児医学奨学財団 研究助成
石塚佳奈子
Authorship：Principal investigator Grant type：Competitive
-
ミクログリア特異的遺伝子CX3CR1に着目して精神障害の分子病態に迫る
2017.08 - 2018.07
特定医療法人万成病院小林孫兵衛記念医学振興財団 研究助成
石塚佳奈子
Authorship：Principal investigator Grant type：Competitive
Other External Funds
-
AYA世代の精神疾患高リスク群における予防的睡眠マネジメントに関する研究
2020.04 - 2021.04
国立研究開発法人日本医療研究開発機構（AMED） 令和2年度 障害者対策総合研究開発事業
水野雅文、石塚佳奈子(分担)
Grant type：Competitive
-
NRXN1を起点に自閉スペクトラム症と統合失調症をgenotype-to-phenotypeで再構築する試み
2018.04 - 2019.03
川野正登記念（公財）川野小児医学奨学財団 研究助成
石塚 佳奈子、石塚佳奈子
Grant type：Competitive
-
ミクログリア特異的遺伝子CX3CR1に着目して精神障害の分子病態に迫る
2017.08 - 2018.07
特定医療法人万成病院小林孫兵衛記念医学振興財団 研究助成
石塚 佳奈子、石塚佳奈子
Grant type：Competitive
Committee Memberships
-
日本精神神経学会/専門医・指導医 代議員
2021.04
Committee type：Academic society
-
日本精神神経学会 代議員
2021.04
Committee type：Academic society
-
日本児童青年精神医学会/認定医・子どものこころ専門医 「児童青年精神医学とその近接領域」編集委員
2020.10
Committee type：Academic society
-
日本児童青年精神医学会 「児童青年精神医学とその近接領域」編集委員
2020.10
Committee type：Academic society
-
臨床精神薬理 編集協力委員
2019.01
Committee type：Academic society
Social Activities
-
名古屋市教育委員会特別支援教育スーパーバイザー
Role(s)： Advisor
2017.05 - 2021.03
-
豊田市こども発達センター児童精神科
Role(s)： Advisor
2012.04
Audience： Infants, Schoolchildren, Junior students, High school students, College students
Type：Internet
-
豊田市教育委員会児童精神相談員
Role(s)： Advisor
2010.04 - 2016.03
Audience： Infants, Schoolchildren, Junior students, High school students
Type：Other
-
西三河児童相談所
Role(s)： Advisor
2010.04 - 2014.03
Audience： Infants, Schoolchildren, Junior students, High school students
Type：Other
-
名古屋市昭和保健所健診事後相談
Role(s)： Advisor
2010.04 - 2012.03
Audience： Infants
Type：Other
-
名古屋市南部地域療育センター
Role(s)： Advisor
2009.04 - 2012.03
Audience： Infants, Schoolchildren, Junior students
Type：Other